Health condition · Clinically reviewed
Heart block, from a long PR interval to complete AV dissociation.
Not every heart block needs treatment, but knowing which type you have changes everything. A short guide to first, second and third-degree block and when a pacemaker earns its place.
Why trust this guide
- 01
Clinically reviewed
Written by our editorial team and reviewed by a registered UK clinician before publication.
- 02
Sourced from guidance
Checked against ESC, NICE and Resuscitation Council UK sources you can see at the end.
- 03
Current for 2026
Reflects modern UK practice on pacing, ambulatory monitoring and treatment of the underlying cause.
Key facts
Heart block at a glance.
The essentials, in plain English. What each type means on the ECG, and what it changes in treatment.
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What it is
Impaired electrical conduction from the atria to the ventricles at the AV node or His-Purkinje system.
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First-degree
Prolonged PR interval over 200 milliseconds. Usually benign and no treatment needed.
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Second-degree
Mobitz I (Wenckebach) is usually benign. Mobitz II and high-grade forms carry a higher risk and often need pacing.
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Third-degree
Complete AV dissociation with a ventricular escape rhythm. A medical emergency and a firm pacing indication.
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Common causes
Ischaemic heart disease, conduction system fibrosis (Lenegre-Lev), medications, electrolytes and post-cardiac-surgery.
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Definitive care
Permanent pacemaker for symptomatic or high-risk block, alongside treatment of any reversible driver.
Why this guide matters
The label on the ECG changes the plan.
The classification of heart block is not academic. It tells clinicians which patients can be reassured and which need urgent pacing.
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Not every block is the same
First-degree and Wenckebach are usually benign. Mobitz II, high-grade and complete block are not.
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Reversible causes matter
Medications, potassium, ischaemia and infection can all drive block. Fixing the cause sometimes fixes the rhythm.
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Pacing is safe and effective
For symptomatic and high-risk block, a permanent pacemaker prevents blackouts and restores a normal life.
How the diagnosis is made
From the first ECG to a settled plan.
The steps a UK cardiologist will normally follow, in order, so you know what to expect and why each test earns its place.
Phase 1 · Assessing
History, ECG and reversible causes
Phase 2 · Confirming
Ambulatory monitoring and imaging
Phase 3 · Planning
Electrophysiology and specialist decision
- 01
Assessing
History and symptom review
Fatigue, dizziness, syncope, palpitations and breathlessness plus a medication and family history for channelopathies.
- 02
Assessing
12-lead ECG
The single most important test. Classifies the block and looks for ischaemia, bundle branch disease and QT changes.
- 03
Assessing
Bloods and reversible causes
Potassium and other electrolytes, troponin if ischaemic, thyroid function and infection screen where relevant.
- 04
Confirming
Ambulatory monitoring
Holter or an implantable loop recorder captures intermittent block that a single ECG can miss.
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Confirming
Echocardiogram and cardiac MRI
Looks for structural heart disease, cardiomyopathy, infiltration and fibrosis of the conducting tissue.
- 06
Planning
Electrophysiology study
Selective and specialist commissioned. Localises the level of block in equivocal cases.
- 07
Planning
Specialist cardiology review
Cardiology and electrophysiology decide on pacing, device selection and management of any underlying condition.
Typical timeline: from an ECG to a settled plan in days for high-risk block, weeks for slower presentations.
Symptoms
How heart block actually feels.
Anything from nothing at all to sudden blackouts. The features that push the diagnosis from watchful waiting to urgent care.
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Often silent (first-degree)
First-degree block is usually an incidental ECG finding with no symptoms at all.
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Occasional palpitations
Wenckebach (Mobitz I) may cause a sense of dropped or skipped beats, often felt at rest.
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Fatigue and reduced exertion
A slower ventricular rate can leave you tiring quickly on stairs and everyday tasks.
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Dizziness and near-syncope
Presyncope on standing or exertion is a warning sign in Mobitz II and higher-grade block.
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Syncope (blackouts)
Sudden loss of consciousness without warning. See our guide to fainting for context.
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Stokes-Adams attacks
Abrupt syncope with pallor followed by flushing as the heart restarts. A firm indication for pacing.
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Breathlessness and heart failure
A slow rate can trigger congestive symptoms, particularly in older or already-diseased hearts.
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Red flag: cardiac arrest
Prolonged pauses can trigger a cardiac arrest. Any collapse with no pulse needs immediate CPR and 999.
Treatment
How heart block is treated in the UK.
Watchful reassurance for benign forms, emergency stabilisation for complete block, and a permanent pacemaker for high-risk or symptomatic disease.
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First-degree: observe
Usually no treatment. Review any AV-blocking medications and reassure. Follow up if PR is very prolonged or symptoms appear.
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Mobitz I: review and monitor
Rationalise beta-blockers, calcium channel blockers and digoxin. Consider pacing only if syncope or symptoms are attributable.
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Mobitz II and high-grade: pace
Permanent pacemaker is the standard of care. Specialist electrophysiology decides device and lead configuration.
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Complete heart block: pace
A firm pacing indication in almost every case. Emergency stabilisation first, then permanent device.
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Emergency stabilisation
IV atropine, isoprenaline and temporary transcutaneous or transvenous pacing while the underlying cause is treated.
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Treat the underlying cause
Reperfusion for MI, potassium correction for hyperkalaemia, medication review and specialist care for endocarditis or Lyme.
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CRT in selected patients
Cardiac resynchronisation therapy for those with LV dysfunction and pacing needs. Specialist commissioned.
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MDT and lifestyle support
Cardiology, electrophysiology and, where useful, BHF and Cardiomyopathy UK for information and peer support.
For device details, see our guides to pacemaker implantation, implantable loop recorder and TAVR / TAVI. Related infection and structural drivers are covered on our endocarditis and cardiac arrest pages.
What this guide is based on
The sources behind every claim on this page.
UK national guidance and specialist society standards, current at the time of last review.
Key references
Guidelines and standards we relied on.
A quiet reminder
This guide is for information, not medical advice.
Your cardiologist knows your history, your ECG and your medications and can tell you which parts apply to you. If you are unsure, get seen.
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European Society of Cardiology (ESC). Guidelines on cardiac pacing and cardiac resynchronisation therapy.
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NICE. Bradyarrhythmias and clinical knowledge summaries on syncope and heart block.
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Resuscitation Council UK. Adult bradycardia algorithm and peri-arrest care.
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British Heart Foundation and Cardiomyopathy UK. Patient information on heart block and pacemakers.
Red flags
When heart block needs urgent attention.
Some forms can be watched. These are the situations that cannot, and where a cardiology or emergency review is needed.
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Syncope with no warning
A blackout with no prodrome, particularly on exertion, points to a serious rhythm cause and needs urgent cardiology review.
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Stokes-Adams attack
Sudden collapse with pallor and then flushing as circulation returns. A firm indication for admission and pacing assessment.
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Complete (third-degree) block on ECG
Complete AV dissociation with a slow escape rhythm. Treat as an emergency and admit to a cardiac unit.
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Mobitz II
Higher risk of progression to complete block. Almost always needs inpatient pacing assessment even if symptoms are mild.
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New block after inferior MI
The AV node shares its blood supply with the inferior wall. New block after an inferior infarct needs prompt cardiology input.
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Cardiac arrest or prolonged pauses
Pauses over 3 seconds, or a collapse with no pulse, are emergencies. Start CPR and call 999.
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New block after cardiac surgery or TAVI
Conduction disease is a known risk after valve surgery and TAVI. Monitor closely and involve electrophysiology early.
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Infective cause suspected
Fever, murmurs or a tick-bite history raise endocarditis or Lyme carditis. Both need urgent specialist care.
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Severe symptoms on medication
New heart block on beta-blockers, calcium channel blockers, digoxin or amiodarone needs urgent medication review.
Living with it
A treatable rhythm, with a clear ladder.
Four things that make the biggest difference day to day. Know your ECG label, watch your medications, take symptoms seriously and accept a pacemaker if it is offered.
A quiet reminder
A pacemaker is not a limit. It is a licence to live normally.
Modern devices are small and reliable. For people with high-risk block they prevent blackouts and support an ordinary, active life.
- 01 Know
Learn your ECG pattern
Ask your cardiologist which type of block you have and what to watch for. Knowing the label makes information easier to find.
- 02 Meds
Review AV-blocking drugs
Beta-blockers, verapamil, diltiazem, digoxin and amiodarone can all worsen block. Any dose change should be discussed with a clinician.
- 03 Symptoms
Take dizziness seriously
Presyncope, syncope or a new drop in exercise tolerance deserves a same-week review, sooner if blackouts happen.
- 04 Pacemaker
If a pacemaker is offered, accept
Modern devices are small, reliable and largely invisible in daily life. They prevent blackouts and support a normal routine.
Frequently asked
Everything we get asked about heart block.
Quick answers on classification, causes, pacing and what to expect after treatment.
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What is heart block?
Heart block, or AV block, is a delay or failure of electrical signals travelling from the atria to the ventricles. It ranges from a benign prolonged PR interval on the ECG through to complete AV dissociation, which is a medical emergency.
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Is first-degree heart block serious?
For most people, no. It is usually an incidental ECG finding with no symptoms and no treatment needed. Very long PR intervals or those found alongside other conduction disease can still deserve a cardiology review, and any AV-blocking medications should be reviewed.
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What is the difference between Mobitz I and Mobitz II?
In Mobitz I (Wenckebach) the PR interval lengthens progressively until a beat is dropped. It is usually benign. In Mobitz II the PR interval stays constant and beats are dropped without warning. Mobitz II carries a much higher risk of progressing to complete block and almost always needs a pacemaker.
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What causes complete heart block?
Common causes include ischaemic heart disease, fibrosis of the conducting system (Lenegre-Lev), cardiomyopathy, medications, electrolyte disturbance, infection such as endocarditis or Lyme carditis, infiltrative disease such as amyloid or sarcoid, congenital block (including neonatal lupus) and complications of valve surgery or TAVI.
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When is a pacemaker needed?
A permanent pacemaker is standard for symptomatic Mobitz I, Mobitz II, high-grade AV block and complete heart block. The decision is made by a specialist cardiologist or electrophysiologist after ECG, ambulatory monitoring and imaging.
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Can heart block be treated by fixing the cause?
Sometimes. Block from an acute MI, hyperkalaemia, Lyme carditis or a rate-slowing drug can resolve once the underlying problem is corrected. In many other cases, particularly conduction system fibrosis, block is permanent and pacing is the definitive treatment.
Related content
Keep reading.
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Heart arrhythmia
The wider family of rhythm disturbances.
Learn more -
Heart rhythm problems
When and why the heart beats out of time.
Learn more -
Heart palpitations
The feeling of a skipped or pounding beat.
Learn more -
Fainting (syncope)
Common causes and when a blackout is serious.
Learn more -
Heart disease
Coronary and structural drivers of block.
Learn more -
Pacemaker implantation
The definitive treatment for high-risk block.
Learn more -
Catheter ablation and EP study
Related electrophysiology procedures.
Learn more -
Subcutaneous ICD
Device therapy for high-risk arrhythmias.
Learn more -
Implantable loop recorder
Long-term monitoring for intermittent block.
Learn more -
Echocardiogram
Structural imaging to look for a cause.
Learn more -
Cardiac MRI
Fibrosis and cardiomyopathy assessment.
Learn more -
All conditions
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