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Neuromuscular neurology · UK

A myasthenia gravis clinic, by a neuromuscular specialist.

A dedicated private MG service in London: AChR, MuSK and LRP4 serology, single-fibre EMG, thymectomy per MGTX, IVIg and PLEX, and access to eculizumab, ravulizumab, efgartigimod, rozanolixizumab and zilucoplan for refractory disease.

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A radiographer guides a patient onto the bed of an advanced 3 Tesla MRI scanner in a London imaging suite

Why patients choose us

  • 01

    A named neuromuscular neurologist, in a specialist MG clinic

    Not a general neurology list. A consultant with a dedicated myasthenia caseload, day-relay infusion access, and a thoracic surgery link if thymectomy is on the table.

  • 02

    The right serology and neurophysiology, in the right order

    AChR-Ab first, then MuSK and LRP4 if seronegative. Single-fibre EMG in a lab that actually runs SFEMG, not just repetitive nerve stimulation on a bad day.

  • 03

    Independent, and free

    We are paid by no clinic, so the introduction is impartial and costs you nothing.

Indicative pricing

What a private MG clinic costs in the UK.

Indicative ranges across our partner units. Send your history and any tests and we quote firm figures across two or three options.

In short

A first neuromuscular consult in London: £350 to £550, workup within two weeks.

Service Indicative range
Initial neuromuscular consult (45 to 60 min) £350 to £550
AChR-Ab, MuSK-Ab and LRP4-Ab serology panel £280 to £520
Single-fibre EMG (SFEMG) or RNS £650 to £1,400
CT chest for thymoma screening £450 to £850
IVIg 2g/kg course (5 day infusion) £4,500 to £9,000
Plasma exchange (PLEX), five sessions £8,000 to £14,000
Thymectomy (VATS or transsternal, per MGTX) £14,000 to £24,000
Eculizumab (Soliris) refractory AChR programme £150,000+ per year

Prices vary by hospital, by consultant, by whether IVIg or PLEX runs as day-case or inpatient, and by biologic funding route. We come back with a firm quote within one working day.

The journey

From referral to biologic, what happens, in order.

One team from first message to long-term monitoring, including infusions and thymectomy planning where indicated.

  1. 01

    Before

    You send us your history and any tests

    A short confidential form: fluctuating weakness, ptosis, diplopia, bulbar or respiratory symptoms, and any prior serology, EMG or CT.

  2. 02

    Before

    We come back with a recommendation

    Within one working day: whether you need urgent review, a neuromuscular clinic, or a second opinion. Indicative price. An honest read either way.

  3. 03

    Before

    We arrange the consultation and workup

    Usually within one to two weeks. AChR-Ab, MuSK-Ab, LRP4-Ab, SFEMG or RNS, ice pack test if ocular, and CT chest to exclude thymoma.

  4. 04

    On the day

    Clinic assessment

    A 45 to 60 minute consult: MGFA classification, Osserman staging where relevant, review of triggers and drugs, and a shared treatment plan.

  5. 05

    On the day

    Treatment started or adjusted

    Pyridostigmine dose optimisation, prednisolone with slow titration, and steroid-sparing IST chosen for your subtype and comorbidities.

  6. 06

    On the day

    Home the same day

    Written plan, safety-net advice on myasthenic crisis, and a 24-hour contact for deterioration between clinic visits.

  7. 07

    After

    Infusions, monitoring and biologics

    IVIg or PLEX arranged if you need bridging. Eculizumab, ravulizumab, efgartigimod, rozanolixizumab or zilucoplan discussed for refractory disease.

When it helps

The presentations we see, and the ones that need A&E.

MG ranges from purely ocular (MGFA I) through generalised (II to IV) to intubated crisis (V). Each has its own pathway.

  • Fatigable ptosis and diplopia

    Drooping eyelids and double vision that worsen with sustained upgaze or through the day. The ice pack test is a bedside pointer for ptosis.

  • Bulbar weakness

    Slurred speech, nasal voice, choking on liquids, difficulty chewing. The MuSK-positive subgroup often presents this way, with respiratory involvement.

  • Generalised limb weakness

    Proximal fatigable weakness in shoulders and hips. Worse after repetition and towards evening. The pattern that turns MGFA class I into class II or III.

  • Respiratory decompensation

    Breathlessness on exertion, orthopnoea, forced vital capacity below 20 ml/kg. Myasthenic crisis needs ITU and IVIg or PLEX, not clinic titration.

  • AChR-positive generalised MG, under 65

    The MGTX trial group where thymectomy adds clear steroid-sparing and functional benefit alongside prednisolone.

  • Thymoma on CT chest

    Around 10 to 15 per cent of AChR-positive patients. Thymectomy is indicated whatever the age or MGFA class.

  • Refractory MG despite two steroid-sparing agents

    Where azathioprine, mycophenolate, methotrexate, ciclosporin or tacrolimus have failed. Complement (eculizumab, ravulizumab, zilucoplan) or FcRn (efgartigimod, rozanolixizumab) are on the table.

  • Red flag: myasthenic crisis

    Rapidly worsening bulbar or respiratory weakness. This is an A&E and ITU situation, not a private booking. Call 999 and go straight in.

Treatment options

Symptomatic, immunosuppressive, surgical and biologic.

Modern MG treatment stacks four layers. Where you sit depends on serology, MGFA class, response so far, and life plans.

  • Symptomatic: pyridostigmine

    The workhorse acetylcholinesterase inhibitor. Titrated 30 to 60 mg every four to six hours, watching for GI cramps, secretions and cholinergic effects.

  • Corticosteroids: prednisolone slow titration

    Started low and increased slowly to avoid an early steroid dip. Often bridged with IVIg or PLEX in more severe disease.

  • Steroid-sparing IST

    Azathioprine, mycophenolate mofetil, methotrexate, ciclosporin or tacrolimus. Choice depends on subtype, comorbidities, fertility plans and TPMT status.

  • IVIg 2g/kg for crisis or bridging

    Intravenous immunoglobulin over two to five days. Rapid onset within a week, effect lasts four to six weeks. Renal function and thrombosis risk reviewed first.

  • Plasma exchange (PLEX)

    Five sessions over roughly two weeks. Fastest onset of any therapy. Vascular access, calcium and clotting all managed on the unit.

  • Thymectomy per MGTX

    VATS or transsternal. Indicated in AChR-positive generalised MG under 65, and for any thymoma. Adds steroid-sparing benefit over years, not weeks.

  • Complement inhibitors

    Eculizumab (Soliris) and ravulizumab (Ultomiris) block C5. Zilucoplan (Zilbrysq) is a daily subcutaneous option. All licensed for refractory AChR-positive MG.

  • FcRn inhibitors

    Efgartigimod (Vyvgart, licensed 2024) and rozanolixizumab (Rystiggo) reduce pathogenic IgG. Cyclical dosing, useful when disease waxes and wanes.

Our vetted London network

A small panel of neuromuscular consultants, we picked them.

Consultants with dedicated MG caseloads in units with infusion capacity and thoracic surgery links. Introductions are made privately, once we understand your case.

  • National Hospital for Neurology (UCLH Private)

  • King’s College Hospital Private Neurology

  • HCA The Wellington Neurosciences

  • Cleveland Clinic London

  • Imperial Private, Charing Cross

  • Consultant neurologists with a dedicated neuromuscular and myasthenia caseload

  • SFEMG and RNS delivered in accredited neurophysiology labs, not general EMG lists

  • Same-week access to IVIg and PLEX, with day-relay infusion capacity

  • Thoracic surgery link for VATS or transsternal thymectomy where MGTX criteria fit

Safety and monitoring

The things worth planning, honestly.

MG is treatable, but the drug avoidance list, IST monitoring and crisis planning matter more than any single prescription.

  • Drug avoidance list

    Aminoglycosides, fluoroquinolones, telithromycin, intravenous magnesium and neuromuscular blockers can precipitate crisis. Beta blockers, macrolides and some statins are used cautiously.

  • Steroid side effects

    Weight, mood, glucose, bone density and cataracts. Bone protection, PPI cover and glycaemic monitoring are set up from day one on prednisolone.

  • IST monitoring

    FBC, LFTs and U&Es every two weeks initially, then monthly. TPMT before azathioprine, drug levels for ciclosporin and tacrolimus.

  • Vaccination and infection risk

    Meningococcal vaccination before eculizumab or ravulizumab. Annual influenza, COVID and pneumococcal cover for all patients on immunosuppression.

  • Myasthenic vs cholinergic crisis

    Sudden worsening of weakness can be either too little or too much acetylcholine. Never guess at home. FVC, bulbar function and cholinergic signs guide the call.

  • Pregnancy planning

    Mycophenolate and methotrexate are teratogenic. Azathioprine, ciclosporin, prednisolone and IVIg are the usual antenatal choices. We plan this before you conceive.

  • Anaesthesia and surgery

    Any planned surgery is flagged to anaesthetics in advance. Suxamethonium and non-depolarising blockers are used with care and reversal is planned.

  • Fatigue and driving

    Diplopia and fatigable weakness affect fitness to drive. DVLA notification is required for generalised MG, and we help you frame it.

  • Red flags after clinic

    Worsening swallowing, breathlessness, orthopnoea, weak cough. Call the 24-hour contact or go to A&E the same day.

Reading your clinic letter

Your MG letter in four parts. Read the last one first.

Whichever consultant sees you, the letter that follows keeps to the same shape.

  1. 01 Header

    Subtype, serology and MGFA class

    AChR-positive, MuSK-positive, LRP4-positive or seronegative. MGFA class I ocular through class V intubated. This drives every downstream decision.

  2. 02 Technique

    Neurophysiology and imaging

    SFEMG jitter and blocking, RNS decrement percentages, ice pack test result, and CT chest findings on the thymus.

  3. 03 Findings

    Triggers and drug review

    Recent infections, missed doses, new prescriptions from the avoidance list, and any procedures on the horizon that need pre-planning.

  4. 04 Impression

    Plan and safety net

    Read this first: pyridostigmine dose, steroid and IST plan, whether IVIg, PLEX, thymectomy or a biologic is on the pathway, and the 24-hour contact.

Recognised by major UK insurers

BupaAXA HealthVitalityAvivaWPACignaHealixBupaAXA HealthVitalityAvivaWPACignaHealixBupaAXA HealthVitalityAvivaWPACignaHealix

Cover for MG varies by insurer. Consults, workup and IVIg or PLEX are usually funded when medically indicated. Biologics need a funding review.

Frequently asked

Everything we get asked about MG.

Quick answers on diagnosis, thymectomy, biologics and drug safety.

  • What is myasthenia gravis?

    MG is an autoimmune disease where antibodies against the postsynaptic acetylcholine receptor (AChR), or against MuSK or LRP4, block neuromuscular transmission. The result is fluctuating fatigable weakness of ocular, bulbar, limb or respiratory muscles. It is treatable, and most patients do well with a well-run plan.

  • How is MG diagnosed?

    By clinical pattern (fatigable weakness, ptosis, diplopia), serology (AChR-Ab, then MuSK-Ab and LRP4-Ab if seronegative), and neurophysiology (single-fibre EMG for jitter and blocking, or repetitive nerve stimulation for decrement). The bedside ice pack test supports ocular MG. Every AChR-positive patient has a CT chest to exclude thymoma.

  • What does private treatment cost?

    A first consult is roughly £350 to £550. Serology and neurophysiology add £1,000 to £2,000. An IVIg 2g/kg course is £4,500 to £9,000. PLEX runs £8,000 to £14,000. Thymectomy is £14,000 to £24,000. Eculizumab is over £150,000 per year and needs a funding route.

  • Should I have a thymectomy?

    The MGTX trial supports thymectomy in AChR-positive generalised MG under 65, alongside prednisolone. Any thymoma is a separate, absolute indication. Video-assisted (VATS) is preferred where anatomy allows, otherwise transsternal. MuSK-positive patients are usually not thymectomised.

  • When are biologics like eculizumab or efgartigimod used?

    For refractory generalised MG despite two steroid-sparing agents and either IVIg or PLEX. Eculizumab and ravulizumab (complement C5), and zilucoplan (subcutaneous C5), are for AChR-positive disease. Efgartigimod and rozanolixizumab (FcRn inhibitors) reduce pathogenic IgG and suit patients with waxing-and-waning disease.

  • Which drugs should I avoid?

    Aminoglycosides, fluoroquinolones, telithromycin, intravenous magnesium and neuromuscular blockers can precipitate a crisis. Beta blockers, macrolides, some statins and certain antimalarials are used cautiously. Always tell every prescriber that you have MG, and carry a card.

Ready to be seen

A neuromuscular consultant, within two weeks.

Send your history and any prior serology. We come back with a named consultant, a workup plan, and firm pricing within one working day.

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